Anti-wrinkle injections, as the name suggests, is a treatment which promotes a smoother and more youthful appearance. It consists of a series of injections, which are used to relax the muscles that cause wrinkles and fine lines by repeated muscle movement.
Botulinum toxin is a purified substance that's derived from bacteria. Injections of botulinum toxin block the nerve signals to the muscle in which it was injected. Without a signal, the muscle is not able to contract. The end result is diminished unwanted facial wrinkles or appearance.
Botulinum toxin can be combined with other cosmetic skin procedures—such as chemical peels, laser treatments, dermal fillers or microdermabrasion—to further improve your results. This combination of therapies can even help to prevent the formation of new lines and wrinkles.
This neurotoxin is produced by the bacterium Clostridium botulinum. C. botulinum elaborates eight antigenically distinguishable exotoxins (A, B, C1, C2, D, E, F and G). All serotypes interfere with neural transmission by blocking the release of acetylcholine, the principal neurotransmitter at the neuromuscular junction, causing muscle paralysis.
The weakness induced by injection with our botulinum toxin usually lasts about six to nine months. Botulinum toxins now play a very significant role in the management of a wide variety of medical conditions, especially strabismus and focal dystonias, hemifacial spasm, and various spastic movement disorders, headaches, hypersalivation, hyperhidrosis, and some chronic conditions that respond only partially to medical treatment.
The cosmetological applications include correction of lines, creases and wrinkling all over the face, chin, neck, and chest to dermatological applications such as hyperhidrosis. Injections are generally well tolerated and side effects are few. A precise knowledge and understanding of the functional anatomy of the mimetic muscles is absolutely necessary to correctly use botulinum toxins in clinical practice.
The toxin requires 24 to 48 hours to take effect, reflecting the time necessary to disrupt the synaptosomal process. In very rare circumstances, some individuals may require as many as 3 to 5 days for the full effect to be observed.
